The event was held live on January 15, 2026. Watch the full recording on demand.
Mitochondria–lysosome crosstalk functions as a core signaling axis regulating immune cell metabolism, stress adaptation, and cell fate, mediated by Ca²⁺ flux, ATP–mTORC1 signaling, and ROS at organelle contact sites. Disruption of this crosstalk contributes to neurodegeneration, metabolic disorders, and cancer, while T-cell–focused studies reveal critical effects on Treg fitness, CD8⁺ T-cell effector function, immune activation, and PANoptosis, supported by advanced tools for monitoring mitochondrial health, and inflammatory signaling, in immunometabolism research.
In this webinar, Dr. Eqra Hafeez shared expert perspectives on:
● Organelle contact sites as immune signaling hubs
● Ca²⁺, ROS, and mTORC1 integrate metabolic and stress signals
● Mechanistic regulation of Treg and CD8⁺T-cell metabolism
● Insights into neurodegeneration, cancer, and metabolic disease models
● Validated tools for mitochondrial and lysosomal health
For inquiries: events@elabscience.com
Speaker
Dr. Eqra Hafeez
Business Development
Elabscience Bionovation Inc.
Dr. Eqra Hafeez is a biologist with a research focus on the cellular and molecular mechanisms of aging, stress resistance, and metabolism. Her recent Ph.D. work investigated the regulation of aging in Drosophila melanogaster, employing advanced techniques in transcriptomics and metabolomics to understand intracellular signaling and organelle function. With a strong background in immunochemistry, enzyme assays, and molecular biology, her expertise provides a valuable perspective on the crosstalk between critical organelles like mitochondria and lysosomes. Dr. Hafeez brings additional experience as a former Lecturer in Zoology and Research Assistant, contributing a broad biological foundation to the discussion of immune cell fate and function.
