Thursday, Feb 26, 2026
11:00 CET (Berlin, UTC+1) | 14:00 GST (Dubai, UTC+4) | 15:30 IST (New Dehli, UTC +5:30) | 05:00 EST (New York, UTC-5)
T regulatory (Treg) cells, driven by Foxp3, are central to immune tolerance, orchestrating immunosuppressive mechanisms that regulate T-cell activation, control inflammation, and maintain tissue integrity. Dysregulation of Treg development or function contributes to autoimmune diseases, cancer immune evasion, and metabolic disorders. Recent breakthroughs in Treg biology, recognized as strong candidates for the 2025 Nobel Prize, reveal cellular and molecular mechanisms that provide critical insights for designing targeted immunotherapies and innovative strategies to restore immune homeostasis.
Key Takeaways:
● Mechanistic insights into Treg cell biology and Foxp3 function
● Cellular and molecular bases of Treg-mediated immunosuppression
● Role of Treg cells in disease pathogenesis and therapy
● Advanced detection tools and analytical approaches for Treg cells
● Receive a digital participation certificate
Who Should Attend:
● Immunologists & biophysicists
● Molecular & cell biologists
● Researchers, and students interested in Treg biology and translational immune regulation
Join Us:
Register today to discover Nobel Prize–inspired insights into Treg cells and their translational impact on immune regulation.
For inquiries: events@elabscience.com
Speaker
Dr. Wang Ying
Product Manager
Elabscience Bionovation Inc.
Dr. Wang Ying is a biophysicist specializing in metabolic diseases and physical intervention therapies. Her doctoral research focused on multi-scale tissue, cellular, in vitro, and transcriptomic approaches to show that static magnetic fields enhance metabolic organ function by promoting actin polymerization, autophagy, and immune cell polarization, thereby improving diabetes and its complications. With expertise in cellular and molecular biology and interdisciplinary analysis, she brings a mechanistic perspective on cell behavior and signaling networks.
