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Purified Anti-Mouse CD200 Antibody[OX-90], Functional Grade (E-AB-F12340)

Purified Anti-Mouse CD200 Antibody[OX-90], Functional Grade - 1
  • Purified Anti-Mouse CD200 Antibody[OX-90], Functional Grade - 1
  • Purified Anti-Mouse CD200 Antibody[OX-90], Functional Grade - 2
All Size Price Qty
100μg $ 50.00
- +
25μg $ 20.00
- +
100mg $ 4100.00
- +
50mg $ 2850.00
- +
25mg $ 1900.00
- +
5mg $ 570.00
- +
1mg $ 156.00
- +
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For research use only.

Conjugation: None (AF/LE)

APC Elab Fluor®700 FITC None (AF/LE) PE/Cyanine 7 Unconjugated Show More

Inquire about other conjugation options

Verified Samples Verified Samples in FCM: C57/BL6 Mouse splenocytes
Dilution FCM 2 μg/mL(0.5×106-1×106 cells),  Block Block- Reported in the literature
Isotype Rat IgG2a, κ
Host Rat
Reactivity Mouse
Applications FCM,  Block
Clonality Monoclonal
Immunogen Recombinant Mouse CD200 protein
Abbre CD200
Synonyms Mox,  OX2,  Mox2,  MRC,  OX-2,  OX-2 membrane glycoprotein,  Cd200
Swissprot
Cellular Localization Cell membrane
Concentration ≥ 1 mg/mL
Buffer Sterile PBS, pH 7.2. < 1.0 EU per mg of the antibody as determined by the LAL method.
Purification Method >98%, Protein A/G purified
Clone OX-90
Conjugation None (AF/LE)
Storage Store at 4°C valid for 12 months or -20°C valid for long term storage, avoid freeze / thaw cycles. This preparation contains no preservatives, thus it should be handled under aseptic conditions.
Shipping Ice bag
background CD200, also known as OX-2, is a 45 kDa transmembrane immunoregulatory protein that belongs to the immunoglobulin superfamily (1, 2). The human CD200 cDNA encodes a 278 amino acid (aa) precursor that includes a 30 aa signal sequence, a 202 aa extracellular domain (ECD), a 27 aa transmembrane segment, and a 19 aa cytoplasmic domain. Costimulates T-cell proliferation. May regulate myeloid cell activity in a variety of tissues (By similarity). None (Azide-Free,Low Endotoxin) are perfectly suited to be used in culture or in vivo (for nonhuman studies) for functional assays blocking, neutralizing, activation or depletion where the presence of azide may damage cells or exogenous endotoxin may signal or activate cells.
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